Evolution: Mimicry meets the mitochondrion
نویسندگان
چکیده
منابع مشابه
Evolution: Mimicry meets the mitochondrion
One hundred and thirty-five years ago, Henry Walter Bates noticed striking similarities between unrelated pierid, ithomiine and heliconiine butterflies collected from the same area [1]. Bates termed this phenomenon ‘mimicry’, and showed that rare or unprotected butterflies would gain by imitating the warning patterns of commoner, unpalatable forms. Later, Müller [2] showed that unpalatable spec...
متن کاملThe Evolution of Mimicry
Cryptic species have evolved camouflage, which enhances survival by decreasing their visibility and thus protecting them from would-be predators. Conversely, aposematic species have evolved vibrant colors which enhance visibility. These warning signals work by helping unpalatable, toxic, evasive, or stinging prey stand out from more favorable prey. Thus, predators learn to generalize the appear...
متن کاملEvolution and disease converge in the mitochondrion.
Mitochondrial DNA (mtDNA) mutations are long known to cause diseases but also underlie tremendous population divergence in humans. It was assumed that the two types of mutations differ in one major trait: functionality. However, evidence from disease association studies, cell culture and animal models support the functionality of common mtDNA genetic variants, leading to the hypothesis that dis...
متن کاملModeling the Evolution of Mimicry
A novel agent based, artificial life model, for the evolution of mimicry is presented. This model is a predator-prey coevolution scenario where pattern representation phenotype is simulated with Cellular Automata (CA), while behaviors of pattern recognition is configured with Hopfield Network. A visual three dimensional toroidal cube is used to construct a universe in which agents have complete...
متن کاملDynamics of mimicry evolution
We simulated mimicry evolution by allowing three populations to coevolve: two populations of senders and one of receivers. Artificial neural networks were used to model receivers, and it was assumed that recognition was inherited. The senders’ signals consisted of nine dimensions. Changes to receivers and senders were caused by random mutations during the course of the simulation. Whereas it pa...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: Current Biology
سال: 1996
ISSN: 0960-9822
DOI: 10.1016/s0960-9822(02)00631-0